Botanical Breast Enhancement: Guide

Biology

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Breast development

The hormones of estrogens, progestogens, prolactin and androgens influence breast tissue through Estrogen Receptor Alpha (ERα), Progesterone Receptor B (PRB), and Prolactin Receptor (PrlR). Estrogen Receptor Beta (ERβ) in the reproductive tract is important for estrogen balance. As each receptor is positively stimulated by its respective hormone, it also becomes desensitized. There are more receptor types in the breast that cannot be ignored due to health reasons, but those mentioned above regulate breast tissue.

Positive estrogenic stimulation, or agonism, of ERα causes lengthening of milk ducts. Branching of milk ducts, which increases the amount of end buds, is caused by progestogenic agonism on PRB. The initial formation of milk lobules converted from the end of milk ducts and their continued growth is caused by prolactin’s effects on PrlR. Progesterone also has a role in differentiation, or conversion of end points into milk lobules, by influencing prolactin, during secretory phase

Endocrinology

Of ERα, PRB, and PrlR, mild potencies of their non-respective hormone enhances each hormone receptor's response to its respective hormone, known as receptor upregulation. Without this synergistic action, the response to a receptor's own specific hormone dulls with quantity or potency, known as receptor downregulation. Too much of a potent hormone may possibly damage its own and other interacting receptors. An imbalance of too much of one type of hormone is a cancer risk. The breast contains more types of cell receptors, but the mentioned above are the focus here.

Estrogens are formed from androgens through a process called aromatase, and aromatase enzymes are located within tissue where ERα and ERβ are also present. This includes ovarian, egg, bone, brain and adipose tissue. Function of theca cells, which produce androgens, and granulosa cells, which produce estrogens, within the follicle or corpus luteum are together important for reproductive health.

Menstrual cycle

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The menstrual cycle will be divided into 5 phases to simply timing: menstruation, proliferative, ovulation, secretory and premenstrual. During menstruation, follicle stimulating hormone (FSH) increases menstruation intensity, and prolactin decreases menstruation intensity. Estrogen is not the dominant hormone for menstruation. During proliferative phase, estrogen levels rise, which develops the egg, and prepares the reproductive tract for it. Estrogen suppresses Luteinizing Hormone (LH) at first, but a buildup of estrogen eventually causes the body to release LH. LH allows ovulation to occur, releasing the egg from the ovary, leaving behind the corpus luteum. The sequential rise of LH, then FSH initially matures the corpus luteum within the ovary during ovulation. FSH then pushes the egg towards the uterus. Progesterone is produced by the corpus luteum, which is a temporary organ whose function is to signal the pituitary gland to momentarily prevent menstruation, for purposes of maintaining fertilization or pregnancy. Lower amounts of estrogens than progestogens are produced during the luteal phase and pregnancy. The pituitary gland releases prolactin, which signals the corpus luteum (and if during pregnancy, the placenta) to release more progesterone, creating a feedback loop. Progesterone increases prolactin, and prolactin lowers FSH and LH. If the egg is not fertilized, the corpus luteum withers within the ovaries. Once progesterone levels drop during premenstrual phase, the pituitary gland signals the release of FSH, allowing menstruation to begin. Outside of the secretory phase, premenstrual phase or pregnancy, progesterone amounts in the body are existant (due to the adrenal glands), but negligible.

About

This is the outdated version of chapter 1 from breast-endocrinology.pdf. The next chapter is Hormone Imbalances. super-bazongas.pdf continues on herb use.

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Breast enhancement; health blog

Blog updates: breast topics and health related content.

Anon05 - August 06, 2019
Breast enlargement herb program for Anon05. Pictures.

Canadian Belle - July 27, 2019
Breast enlargement herb program for Canadian Belle.

Super Bazongas: Upcoming update - August 31, 2019
Adjustments to proliferative phase, to limit herb dose containing fenugreek to once. This suggestion was in a recent past herb schedule. The results have been inconsistent as proliferative phase requires more precise use of herbs, and it is more difficult to find a suitable herb combination than for other phases. Other tweeks to herb schedule.

Silybum; milkthistle - August 30, 2019

Making herbal tea - August 13, 2019
Tips on making herbal tea.

Fennel - August 01, 2019

Anti-mullerian hormone - July 06, 2019
High levels of anti-mullerian hormone (AMH) is associated with high LH and high androgens for those with PCOS. AMH levels are measured to indicate potential fertility. Lowered fertility likely has to do with reduced FSH and estrogen sensitivity.

Anethole & estragole - June 25, 2019
Many known stilbenes are estrogen receptor antagonists. Stilbenes and their polymers include: anethole, dianethole, estragole, tamoxifen, stilbene and stilbestrol. Spearmint and fennel contain stilbenes as active hormonal constituents. Based on anecdotal information from spearmint's and fennel's actions, anethole, dianethole and estragole are likely ERα selective. Anethole is another name for transanethole, and Stilbene is another term for transtilbene. Estragole and anethole are isomers. Dianethole and photoanethole are polymers of anethole.

Fenugreek - June 25, 2019

Evening primrose suggestion removed from proliferative phase - June 19, 2019

Updates - June 04, 2019
Breast and hip tissue should increase the ability for estrogen conversion from androgens. The term hormone receptor blockers is inaccurate to describe hormones' effects according to this research. Phytohormones can multiply, act directly on or cause an inverse action of hormone effects or of other phytohormones. A phytohormone's basic actions are agonize, antagonize and influence bodily hormones. These actions desensitize or sensitize receptors, and increase a bodily response. The ultimate effect depends on the presence of other phytohormones and bodily hormones. Caffeine, for instance, can cause an inverse effect of shrinking tissue size, when added to a herb combination that would otherwise cause growth. Update in schedule: sunflower must not be used in the presence of when negative symptoms occur. June 04, 2019

Program blog for herb use - June 04, 2019
Update for anon03's herb use blog.

Sitemap timestamps - June 04, 2019

Papaya, Black walnut - May 27, 2019
Papaya seeds kill intestinal parasites. Black walnut (Juglans nigra) hull as extract or powder also kills worms. Juglans can cause a hormone imbalance with excessive or extended use, so a small amount is recommeneded. There are other herbs that can get rid of intestinal parasites, but papaya seeds and black wallnut hulls are safer and highly effective. Pineapple also breaks down worms. Pumpkin seed only works against certain species of intestinal worms, and it doesn't completely destroy them. Wormwood kills intestinal worms, but it is easily toxic in high doses.

Opensource Materials - May 22, 2019

Breast Development and Endocrinology, 3rd Ed - May 22, 2019
Expanded and clarified details for Breast Development and Endocrinology 3rd edition. From now on, this will be volume 1. These edits will reflect in Super Bazongas. Herb schedule will be edited, but same herb combinations will be used as basis. Future releases of Super Bazongas will be volume 2.

Vitamin supplementation - April 17, 2019
There has been recent controversy about vitamin supplementation. From now on, suggestions will be limited. Vitamin C will be suggested, as it preserves fat soluble vitamin E, and possibly other fat soluble vitamins and antioxidants. Vitamin E may be used, but use it sparingly and with foods with fiber. If you take vitamin D, be sure it is in a complex with calcium, magnesium and zinc. Vitamin B12 supplementation (with B vitamins complex) is only for those who don't eat meat, dairy or eggs. See /appendix/nutrition for vitamin amounts and for information on nutritious foods.

Acne - March 19, 2019
Acne is caused by androgens, glucocorticoids, corticotropin and insulin. Androgens trigger the production of sebum from sebaceus glands, which causes acne. Insulin affects the size of sebaceus glands. Unfortunately, hair follicles transform mild androgens into DHT, which hirsutism symptoms often show up after acne symptoms go away. When there's acne, there's potential to use herbs with an estrogen analog to cause breast growth. The problem is, acne caused by herbs has seemed to antagonize one of two estrogen receptors before. If the estrogen simile targets the wrong receptor, the herb won't work, and there's potential for negative effects. It is much better to use the right herb combination, than to try to react by adding herbs later. If the correct herb compliment is taken moments late, that can also cause negative results.

Estrogen imbalance - March 16, 2019

Hirsutism blog split to anon04 - March 12, 2019

Recent updates - February 20, 2019

Receptor Regulation - February 20, 2019

2019 archive - June 04, 2019

2018 archive - December 31, 2018

2017 archive - December 31, 2017

2016 archive - December 31, 2016

References: